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Epigenetics and Nuclear Signaling Chromatin Modifying Enzymes Methylation

Human NSD1 (KMT3B) knockout HEK-293T cell line (ab266306)

Human NSD1 (KMT3B) knockout HEK-293T cell line (ab266306)
  • ChIP - Anti-Histone H3 antibody - Nuclear Loading Control and ChIP Grade (ab1791)
  • ChIP - Anti-Histone H3 antibody - Nuclear Loading Control and ChIP Grade (ab1791)

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Properties

  • Number of cells

    1 x 106 cells/vial, 1 mL
  • Viability

    ~90%
  • Adherent /Suspension

    Adherent
  • Tissue

    Kidney
  • Cell type

    epithelial
  • STR Analysis

    Amelogenin X D5S818: 8, 9 D13S317: 12, 14 D7S820: 11 D16S539: 9, 13 vWA: 16, 19 TH01: 7, 9.3 TPOX: 11 CSF1PO: 11, 12
  • Mycoplasma free

    Yes
  • Storage instructions

    Shipped on Dry Ice. Store in liquid nitrogen.
  • Storage buffer

    Constituents: 8.7% Dimethylsulfoxide, 2% Cellulose, methyl ether
  • Purity

    Immunogen affinity purified
  • Research areas

    • Epigenetics and Nuclear Signaling
    • Chromatin Modifying Enzymes
    • Methylation
    • Epigenetics and Nuclear Signaling
    • Nuclear Signaling Pathways
    • Nuclear Receptors
    • Co-activators/co-repressors
    • Epigenetics and Nuclear Signaling
    • Transcription
    • Other factors
    • Epigenetics and Nuclear Signaling
    • Chromatin Modifying Enzymes
    • Methylation
    • Lysine methylation

Images

  • Sanger Sequencing - Human NSD1 knockout HEK293T cell line (ab266306)
    Sanger Sequencing - Human NSD1 knockout HEK293T cell line (ab266306)
    Homozygous: 22 bp deletion in exon4
  • Cell Culture - Human NSD1 (KMT3B) knockout HEK293T cell line (ab266306)
    Cell Culture - Human NSD1 (KMT3B) knockout HEK293T cell line (ab266306)
    Representative images of NSD1 knockout HEK293T cells, low and high confluency examples (top left and right respectively) and wild-type HEK293T cells, low and high confluency (bottom left and right respectively) showing typical adherent, epithelial-like morphology. Images were captured at 10X magnification using an EVOS M5000 microscope.

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